Thursday, December 4, 2008

New Sickle Cell Anemia Treatment discovered

Thanks to hard work, patience, and scientific pursuit of knowledge and application there seems to be New Hope for Sickle-Cell Anemia Sufferers. According to a report in Science Magazine,
Researchers have discovered the molecular switch for activating the fetal form of hemoglobin—the iron-containing protein in red blood cells that transports oxygen—which could help alleviate the symptoms of genetic blood disorders, including sickle-cell anemia.

Sickle anemia is widely regarded as a Black disease because it affects an estimated 70,000 people (mostly African-Americans) in the U.S. But it in fact peoples from many regions of the world have high incidences of this disease in their populations: Africa, Mediterranean countries (such as Greece, Turkey, and Italy), The Arabian peninsula, India, and Latin America (such as South America, Central America, and parts of the Caribbean).

Sickle-cell anemia can be fatal with most people dying by their mid-40s. When I was a young child, persons with sickle-cell anemia had a life expectancy of 25. Thank goodness for modern medicine. However, it is a high-maintenance disease. Sufferers deal with extreme pain in the legs when their red blood cells sickle and prevent a constant flow of oxygenated blood. Many take blood thinners or have frequent blood transfusions to stem the symptoms. This new treatment technique may by-pass all of that. Inherited forms of anemia may soon be treated by turning on a gene normally active only in the womb, when individuals with sickle cell anemia are asymptomatic. Read more about this new discovery at Scientific American.

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